| Surname, First name |
Research Focus |
Availability |
| Hartl, Ulrich |
Research in Hartl and Hayer-Hartl laboratory focuses on the mechanisms of protein folding and quality control in the cell. Our goal is to reach a comprehensive understanding, at the structural and functional level, of how the machinery of molecular chaperones assists folding through the cooperation of co- and post-translational mechanisms. |
not available |
| Kunz, Hans-Henning |
Analysis of putative cyanobacterial thylakoid ion transport proteins for their role in photosynthesis |
not available |
| Ladurner, Andreas |
Next summer, join us in investigating the intricate link between glucose metabolism, transcriptional control and gene regulation in eukaryotes. Our project focuses on how the sugar-tolerance transcription factor ChREBP and its paralogs directly sense cellular metabolites to drive large changes in gene activity. |
not available |
| Nägele, Thomas |
Our aim is to reveal how photosynthesis and carbohydrate metabolism are stabilised in lavender plants under adverse growth conditions. |
available in 2026 |
| Robles, Maria |
While the circadian clock regulates metabolism, metabolic states, in turn, provide feedback to the circadian clock, modulating its function. We are investigating this molecular crosstalk in peripheral metabolic tissues from mice, employing interaction and spatial proteomics as well as phosphoproteomics. |
available in 2026 |
| Sattler, Michael |
The student will contribute to the cloning of expression constructs, recombinant protein production, and purification, forming the foundation for the structural and functional studies. Through this work, we aim to uncover key aspects of the Grp94 functional cycle, providing insights that are fundamental for understanding ER proteostasis and may ultimately inform future drug discovery efforts targeting Grp94. |
available in 2026 |
| Turck, Christoph W. |
The comparison of the proteome of diseased and healthy tissues and body fluids and the subsequent identification of the proteins that are different from normal in disease are pursued in order to unravel the pathogenesis of disease, to identify therapeutic targets, and to develop diagnostic tests. |
not available |
| Willem, Michael |
Alzheimer’s disease (AD) is the main cause of dementia and a clear challenge for modern public health. Triggering receptor expressed on myeloid cells 2 (TREM2) is expressed by microglia, and sequence variants have been identified as risk factors for AD. The project will address the direct interaction of soluble TREM proteins with candidate receptors using site-directed mutagenesis and functional assays to monitor the effects on the interaction and activity in cell culture and in vitro. |
available in 2026 |
