| Surname, First name |
Research Focus |
Availability |
| Baier, Herwig |
The goal of our research is to understand how neuronal circuits convert sensory inputs into behavioral responses. |
TBA |
| Benakis, Corinne |
Stroke is one of the leading causes of death and mortality worldwide. One of the main risk factors for stroke onset is exposure to environmental toxins. Some of these environmental toxins that affect human health include dioxins and polyaromatic hydrocarbons. Both compounds have a high affinity for the aryl hydrocarbon receptor (AhR) which is ubiquitously expressed in immune cells at barrier sites, including the gut, the lung and meninges. For this project, we will test specific environmental toxins, ligands of AhR, and characterize the immune response and microbial composition before and after exposure to the toxin and evaluate the impact on stroke severity in mice. |
available in 2026 |
| Hartl, Ulrich |
Research in Hartl and Hayer-Hartl laboratory focuses on the mechanisms of protein folding and quality control in the cell. Our goal is to reach a comprehensive understanding, at the structural and functional level, of how the machinery of molecular chaperones assists folding through the cooperation of co- and post-translational mechanisms. |
not available |
| Herms, Jochen |
Physiological function of proteins involved in Neurodegenerative diseases, synaptic failure in Neurodegenerative diseases like Alzheimer’s or Parkinson’s disease (AD,PD), long-term in vivo two-photon imaging of transgenic mice models of AD, neuronal calcium homeostasis in neurodegenerative diseases, high-throughput drug screens for AD and PD. |
not available |
| Klein, Rüdiger |
In our work, we aim to understand the effects of these weight loss drugs on the CeA and to determine whether the CeA is required for their action. |
available in 2026 |
| Kunz, Hans-Henning |
Analysis of putative cyanobacterial thylakoid ion transport proteins for their role in photosynthesis |
not available |
| Ladurner, Andreas |
Next summer, join us in investigating the intricate link between glucose metabolism, transcriptional control and gene regulation in eukaryotes. Our project focuses on how the sugar-tolerance transcription factor ChREBP and its paralogs directly sense cellular metabolites to drive large changes in gene activity. |
TBA |
| Lichtenthaler, Stefan |
We study how Alzheimer’s disease develops in the brain on the molecular and cellular level. The aim of our research is to better understand the disease causes and to develop new diagnostic, therapeutic and preventive approaches. |
TBA |
| Michalakis, Stylianos |
Engineered adeno-associated virus-based vectors for retinal gene therapy - Mechanistic studies on cellular infection, trafficking and transduction |
TBA |
| Misgeld, Thomas |
The Misgeld lab uses in vivo imaging methods (ranging from wide-field time-lapse to two-photon microscopy) to study the development and degeneration of neurons and their processes. |
TBA |
| Nägele, Thomas |
Our aim is to reveal how photosynthesis and carbohydrate metabolism are stabilised in lavender plants under adverse growth conditions. |
available in 2026 |
| Niessing, Dierk |
Our goal is to understand the molecular principles underlying cargo recognition by transport complexes, complex assembly and activation, and eventually complex disassembly after the transport. |
TBA |
| Robles, Maria |
While the circadian clock regulates metabolism, metabolic states, in turn, provide feedback to the circadian clock, modulating its function. We are investigating this molecular crosstalk in peripheral metabolic tissues from mice, employing interaction and spatial proteomics as well as phosphoproteomics. |
available in 2026 |
| Stricker, Stefan H. |
The research aim of the lab is to investigate how cells know which cell type they are and why they never forget. We employ a wide range of CRISPR methods to brain cells to test in vitro and in vivo, which epigenetic marks and gene activities have functional relevance in mediating cell identity or disease phenotypes. |
TBA |
| Willem, Michael |
Alzheimer’s disease (AD) is the main cause of dementia and a clear challenge for modern public health. Triggering receptor expressed on myeloid cells 2 (TREM2) is expressed by microglia, and sequence variants have been identified as risk factors for AD. The project will address the direct interaction of soluble TREM proteins with candidate receptors using site-directed mutagenesis and functional assays to monitor the effects on the interaction and activity in cell culture and in vitro. |
available in 2026 |
